Viral Triggers and Inflammatory Mechanisms in Pediatric Epilepsy

نویسندگان
چکیده

برای دانلود رایگان متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Viral and inflammatory triggers of neurodegenerative diseases.

Here, we synthesize research behind the emerging hypothesis that inflammation--which can result, for example, from viral infections--can initiate and propagate chronic neuronal dysfunction, an event that precedes the clinical onset of many neurodegenerative diseases. Therapeutic approaches that target immunological pathways in the prodromal phase of diseases might decrease the incidence of neur...

متن کامل

O12: Presurgical Evaluation in Pediatric Epilepsy Surgery

لطفاً به چکیده انگلیسی مراجعه شود.

متن کامل

O14: Epilepsy Surgery in the Pediatric Age

لطفاً به چکیده انگلیسی مراجعه شود.

متن کامل

Viral Infections and their Role in Autoimmune Diseases, with Emphasis on Mechanisms and Molecular Interactions

Introduction: The exact cause of most autoimmune diseases is still unknown; however, several factors play a role in causing or exacerbating autoimmune reactions. In addition to environmental factors such as bacterial, parasitic, fungal and viral infections, factors such as genetic characteristics and lifestyle are also included. Infections caused by viruses usually trigger a strong immune respo...

متن کامل

Epilepsy and viral infections.

In Clinical Science Viral infections may cause seizures via several pathogenetic mechanisms. Systemic infections, such as influenza, can lead to metabolic compromise, as well as occasional direct central nervous system (CNS) invasion—even though not usually neu-rotrophic. Although viral infection confined to the meninges rarely causes seizures and does not increase risk for later epilepsy , enc...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

ژورنال

عنوان ژورنال: Molecular Neurobiology

سال: 2018

ISSN: 0893-7648,1559-1182

DOI: 10.1007/s12035-018-1215-5